Yamashita, Y.M.(Yukiko Yamashita)
repair illudin lesion fibroblast excision pathway strain jasper panel nucleotide excision repair syndrome tc-ner synthesis n.g.j polymerase survival xeroderma pigmentosum cockayne syndrome xeroderma post-replication repair exposure nucleotide transcription sensitivity response post-replication illudin lesions dna synthesis level pigmentosum cytotoxicity damage gg-ner culture symbol xp-c cells repair pathways defect mutant c 5ro mcmorri activity cell strains cockayne irofulven agent chicken lymphoma cells hoeijmaker dna polymerase eta cancer hamster replication base excision repair factor chinese replication forks yamaizumi kelner global-genome xp 25ro transcription-coupled ner xp-a cells dna repair xp 21ro recovery non-homologous end-joining group chicken curve mechanism mouse protein uv-induced patient translesion caffeine uv-sensitive ovary rad 18/rad protein ham f 10
1 Most Recent Publications
Anti-tumour compounds illudin S and Irofulven induce DNA lesions ignored by global repair and exclusively processed by transcription- and replication-coupled repair pathways.
Raams, A. Kelner, M.J. Ng, J.M.Y. Yamashita, Y.M. Takeda, S. McMorris, T.C. Hoeijmakers, J.H.J. Jaspers, N.G.J.