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Vennekens, K.
( K. Vennekens)
patient amyloid plaque mutation disease alzheimer vessel brain ncstn flemish group study deposit protein control plaque cores section region frequency dementia figure pathology ad /fl association acta neuropathol analysis antibody ad /fl brains amyloid deposits haplotype van broeckhoven c amyloid angiopathy hchwa-d table van duijn cm broeckhoven precursor cruts m ad /fl patients dutch amyloid precursor protein perivascular angiopathy neuropathol hemorrhage van duinen sg formation subject formic coronal kumar-singh peptide al ajp deposition plaque core dystrophic neurites n-truncated genet immunohistochemistry allele duijn neurol 4 g 8 van harskamp f patients iv -4 scale bars hchwa-d patients control subjects frangione b citrate buffer stroke sample cerebellum alssv neurofibrillary amyloid-laden intron secretase ad patients
2 Most Recent Publications
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The gene encoding nicastrin, a major gamma-secretase component, modifies risk for familial early-onset Alzheimer disease in a Dutch population-based sample
(Article)
Dermaut, B. Theuns, J. Broeckhoven, C. van Sleegers, K. Hasegawa, H. Vennekens, K. Corsmit, E. St George-Hyslop, P.H. Cruts, M. Duijn, C.M. van Broeck, M. van den |
2002-01-01
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Dense-core senile plaques in the Flemish variant of Alzheimer's disease are vasocentric
(Article)
Kumar-Singh, S. Lubke, U. Wang, R. Kros, J.M. Swieten, J.C. van Broeckhoven, C. van Ceuterick, C. Serneels, S. Vennekens, K. Duijn, C.M. van Timmermans, J.P. Martin, J-J. Marck, E. van Cras, P. |
2002-01-01
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