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Martin, S.

( S. Martin)


enzyme mutation individual serum angiotensin activity blood family level angiotensin-converting enzyme serum ace hyper-ace j biol chem angiotensin i-converting enzyme analysis ace gene figure amount culture medium hyper-ace individuals alhenc gelas f increase serum ace activity disease region polymorphism member study angiotensin i ace activity serum ace levels protein nonaffected family members point subject family members hyper-ace phenotype cell surface membrane-bound pressure point mutation sequence cleavage plasma nonaffected heart circulation medium patient result culture substrate dendritic cells position abnormality blood pressure regulation hypertension cell-surface ace expression department ace solubilization serum angiotensin-converting enzyme i-converting 3 nonaffected individuals ace levels cell-associated ace angiotensin-converting enzyme causes ace protein serum angiotensin danilov biochem j renin downloaded disease cornelis kramers ace secretion heart failure domain mrna level february elevation cornelia van duijn




1 Most Recent Publications

Point Mutation in the Stalk of Angiotensin-Converting Enzyme Causes a Dramatic Increase in Serum Angiotensin-Converting Enzyme But No Cardiovascular Disease (Article)
Kramers, C. Danilov, S.M. Adema, G.J. Boomsma, F. Deinum, J. Balvasnikova, I.V. Scharenborg, N. Looman, M. Keijzer, M.B. de Duijn, C.M. van Martin, S. Soubrier, F.
2001-01-01