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Esveldt-van Lange, R.
(Rebecca Esveldt-van Lange)
−/− es cells repair recombination mutation end-joining damage apoptosi microhomology use microhomology xpa −/− dna damage response pathway ligase genome tc-ner assay r e p i r 7 product strand protein dna ligase iv type es cells xpc −/− csb −/− cycle dna repair hamster cell lines defect apoptotic level mutant ficient apoptotic response result gg-ner sensitivity broblast chinese fi broblasts cell type genotoxic survival induction microhomology-directed end-joining substrate treatment van der horst philips tuv lamp rag 2 proteins lesion nucleotide excision repair hypersensitivity mechanism uv-induced excision reaction exposure cell cycle 1998. agent table dna double-strand rag 1 van zeeland speci ficiency bstxi wild-type fragment uv-light cell cycle block mouse v-h 4 genotoxic stress bstxi digestion block
2 Most Recent Publications
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Cell-type-specific consequences of nucleotide excision repair deficiencies: Embryonic stem cells versus fibroblasts
(Article)
Waard, H. de Sonneveld, E. Wit, J. de Esveldt-van Lange, R. Hoeijmakers, J.H.J. Vrieling, H. Horst, G.T.J. van der |
2008-10-01
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Different types of V(D)J recombination and end-joining defects in DNA double-strand break repair mutant mammalian cells.
(Article)
Verkaik, N.S. Esveldt-van Lange, R. Heemst, D. van Brüggenwirth, H.T. Hoeijmakers, J.H.J. Zdzienicka, M.Z. Gent, D.C. van |
2002-01-01
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