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scopus: 8543745700

Lintel Hekkert, M. te

(Maaike te Lintel Hekkert)


preconditioning postconditioning heart adenosine pressure stimulus artery infarct size occlusion cardioprotection reperfusion blood protection ischemic preconditioning study myocardium ischemic tolerance infarct effect ischemia 2 cao 15 pressure perturbation artery occlusion volume pathway physiol perturbation preconditioning stimulus aortic 4 cao 15 stimuli 1 ipc 15 3 poc 30 group animal baseline cardiol reactive hyperemia index ischemia ipc stimulus 60- min cao verdouw pd duncker dj duncker development 0.05 period cao 60 patient exogenous adenosine control hyperemia heart rate index infarction episode vivo rat heart signal transduction pathway contraction 3 ipc 3 analysis protocol pressure wave cao 30 increase rat heart 15 min 0.05 vs rabbit 3 cao 3 60- aortic pressure perturbation aortic pressure curve activation reactive verdouw blood pressure result 15-




3 Most Recent Publications

Coronary-aortic interaction during ventricular isovolumic contraction (Article)
Houwelingen, M.J. van Merkus, D. Lintel Hekkert, M. te Dijk, G. van Hoeks, A.P.G. Duncker, D.J.G.M.
2011-04-13
Interaction between pre- and postconditioning in the in vivo rat heart (Article)
Manintveld, O.C. Lintel Hekkert, M. te Ploeg, N.T. van der Verdouw, P.D. Duncker, D.J.G.M.
2009-11-01
Myocardium tolerant to an adenosine-dependent ischemic preconditioning stimulus can still be protected by stimuli that employ alternative signaling pathways. (Article)
Liem, D.A. Lintel Hekkert, M. te Manintveld, O.C. Boomsma, F. Verdouw, P.D. Duncker, D.J.G.M.
2005-03-01