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study patient analysis linkage no-mi panobinostat score chromosome family position genet sample model association phase ketoconazole locus evidence inhibitor phenotype region linkage analysis result disease cyp 3a inhibitor adipor sibling extension phase adipor 2 table core phase allele recessive model slc 4a treatment modi fier genes signi genotype marker method fi brosis greenberg mutation cancer lod score analysis lod score adipor 2 gene brosi trait lod scores recessive cf patients increase period linkage evidence identi hodge pharmacokinetic heterogeneity multipoint value cyp 3a analyses npl analysis mi phenotype hypophosphatemia cyp 3a inhibitors extension phosphate lung disease cf siblings tumor signi ficantly no-mi phenotype genotypic individual canadian interaction pancreatic university
2 Most Recent Publications
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Effect of ketoconazole-mediated CYP3A4 inhibition on clinical pharmacokinetics of panobinostat (LBH589), an orally active histone deacetylase inhibitor
(Article)
Hamberg, P. Woo, M.M. Jonge, M.J.A. de Chen, L.C. Verweij, J. Porro, M.G. Zhao, L. Li, W. Biessen, D. van der Sharma, H.S. Hengelage, T. |
2011-06-27
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Modifier gene study of meconium ileus in cystic fibrosis: Statistical considerations and gene mapping results
(Article)
Dorfman, R. Li, W. MacEk, M. Czerska, K. Sands, D. Tiddens, H.A.W.M. Margarit, S. Repetto, G. Sontag, M.K. Accurso, F.J. Blackman, S. Cutting, G. Sun, L. Tsui, L.C. Corey, M. Durie, P. Zielenski, J. Strug, L.J. Lin, F. Wang, Y. Sandford, A. Paré, P.D. McKay, K. Kayserova, H. Piskackova, T. |
2009-12-01
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