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Tanke, H.J.
( H.J. Tanke)
cancer tumor adenocarcinoma chromosome prostate cancer prostate cardia study esophagus chromosomal number tumor volume esophageal genomic hybridization stage region barrett hybridization amplification analysis volume change alteration prostatic table ptrend screening genomic junction carcinoma patient 8 p loss cytogenetic group chromosomal alterations esophageal adenocarcinoma deletion van dekken h european randomized study frequency rotterdam 14 q 3132.1 tumor stage parameter gej adenocarcinomas figure esophageal adenocarcinomas /or 8 q clinicopathological parameters 11.2-pter progression dekken activation chromosomal regions erspc 11.2-p oncogene sequence gastroesophageal cardia cancers preoperative prostatic tumorigenesis increase allelic loss candidate genes bostwick dg neoplasm 5 p 14 prostatic tumor development grade 1997. 8 q sequences genes chromosomes cancer candidate 19 q 13.1. tv 0.5 ml correlation suppression clinicopathological gleason
2 Most Recent Publications
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Molecular cytogenetic analysis of prostatic adenocarcinomas from screening studies : early cancers may contain aggressive genetic features
(Article)
Alers, J.C. Krijtenburg, P.J. Vis, A.N. Hoedemaeker, R.F. Wildhagen, M.F. Hop, W.C.J. Schröder, F.H. Tanke, H.J. Dekken, H. van Kwast, Th.H. van der |
2001-01-01
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Comparative genomic hybridization of cancer of the gastroesophageal junction: deletion of 14Q31-32.1 discriminates between esophageal (Barrett's) and gastric cardia adenocarcinomas
(Article)
Dekken, H. van Geelen, E. Dinjens, W.N.M. Wijnhoven, B.P.L. Tilanus, H.W. Tanke, H.J. Rosenberg, C. |
1999-01-01
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