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kidney figure heart wild-type receptor animal mitochondrial effect peptide expression control ant /ren rats activity ant 1 overexpression nf-kb germany overexpression natriuretic pressure increase function ren rats hypertrophy disease swets subscription service group wild-type controls c-type natriuretic peptide study wild-type mice protein analysis angiotensin apoptosi inflammation model volume ang ii receptors infarction transgene i /r injury / european journal fibrosis change proinflammatory number staining mas-deficient mice downloaded mitochondria activation cardiomyocyte cytosolic mas-deficient infusion 5 days pathomorphological changes circulation saline failure factor i /r kidneys assay walther ureteral obstruction infarct ant 1 expression kinase contralateral over-expression creatine result damage antibody 7 days nf-kb activity mas +/+ mas receptor 6–10 animals nucleotide
3 Most Recent Publications
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Angiotensin-(1-7) and the G protein-coupled receptor Mas are key players in renal inflammation
(Article)
Esteban, V. Heringer-Walther, S. Walther, T. Sterner-Kock, A. Bruin, R.W.F. de Engel, S. van den Wang, Y. Mezzano, S. Egido, J. Schultheiss, H.P. Ruiz-Ortega, M. |
2009-04-30
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Cardiomyocyte-restricted over-expression of C-type natriuretic peptide prevents cardiac hypertrophy induced by myocardial infarction in mice
(Article)
Wang, Y. Waard, M. de Sterner-Kock, A. Stepan, H. Schultheiss, H.P. Duncker, D.J.G.M. Walther, T. |
2007-06-01
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Accelerated mitochondrial adenosine diphosphate/adenosine triphosphate transport improves hypertension-induced heart disease
(Article)
Walther, T. Tschöpe, C. Bader, M. Shakibaei, M. Schultheiss, H.P. Dörner, A. Sterner-Kock, A. Westermann, D. Heringer-Walther, S. Riad, A. Nikolic, A. Wang, Y. Ebermann, L. Siems, W.-E. |
2007-01-01
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