Cavagnini, F.

ifn -β subject effect treatment type i ifns mutation disease patient expression study protein bxpc -3 cells cancer tumor family growth aip mutations combination endocrinol ifn -α prevalence activation hormone metab receptor group syndrome activity baseline control interferon concentration p 27 expression phosphorylation secretion increase analysis *** factor cerebrovascular disease inhibition vitale pathway adenomas stat -3 incidence therapy department outcome deficiency cerebrovascular 3- yr incidence gh deficiency endocrinology pancreatic signi response experiment type i interferons pancreatic cancer cells binding fipa families fipa cohort cycle cushing acromegaly cortisol ifnar signi ficantly prolactin macro result autophagy mechanism survival troglitazone france van koetsveld pm 3- yr prevalence eif 4e phosphorylation