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neuron motor motor neurons disease g 1del mice protein inclusion golgi dynein amyotrophic expression sclerosis degeneration mouse golgi fragmentation mutation fragmentation interneuron motor neuron degeneration section aggregate pathology model sod 1-als mice sod 1 golgi apparatus transport /dynactin astrocyte bn 1 mice level glyt 2 axonal chapter function journal onset motor neuron disease t 3t mice antibody analysis -43 abnormality gfp-bicd study atf 3 expression vapb inclusions dynein /dynactin function ubiquitinated staining dendrite muscle ubiquitin dynein /dynactin patient aggregation cause ventral horn apparatus cord sections microglia axonal transport neurodegenerative superoxide change confocal non-transgenic neurosci number jaarsma dismutase figure sod 1 aggregates disease progression mechanism stress structure pathway science control
3 Most Recent Publications
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The role of inclusions in ALS pathogenesis
(Doctoral Thesis)
Dis, V. van |
2012-09-19
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Spinal inhibitory interneuron pathology follows motor neuron degeneration independent of glial mutant superoxide dismutase 1 expression in SOD1-ALS mice
(Article)
Hossaini, S.M. Cano, S.C. Dis, V. van Haasdijk, E.D. Hoogenraad, C.C. Holstege, J.C. Jaarsma, D. |
2011-08-01
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A novel mouse model with impaired dynein/dynactin function develops amyotrophic lateral sclerosis (ALS)-like features in motor neurons and improves lifespan in SOD1-ALS mice
(Article)
Teuling, E. Dis, V. van Wulf, P. Haasdijk, E.D. Akhmanova, A. Hoogenraad, C.C. Jaarsma, D. |
2008-09-08
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