Scholte, H.R.

(Hans Scholte)

Supervisor (promotor) of 3 dissertations

patient activity control blood mitochondrial syndrome protein assay mutation study fatigue syndrome sample author spier analysis result article level concentration fatigue i activity leigh syndrome treatment disease group department leigh ammonia plasma exercise 25- defect oxidative phosphorylation assembly spieren work rate cause genet pediatrics oxidative difference i assembly medicine oxygen 20orf hyperinsulinism c 20orf gene mmp -7 rotenone phosphorylation erasmus rotterdam oxidase glutamine duchenne family dehydrogenase journal cfs /me patients tabel bindweefsel vitamin blood ammonia levels symptom laboratory november glutamate editor reaction kinase hyperammonemia netherland liver neoplastin wordt downloaded c 20orf synthesis cpet 1 tissue synthetase

5 Most Recent Publications

Patients with chronic fatigue syndrome performed worse than controls in a controlled repeated exercise study despite a normal oxidative phosphorylation capacity (Article) Vermeulen, R.C.W. Kurk, R.M. Visser, F.C. Sluiter, W. Scholte, H.R.	2010-10-11
Defective complex I assembly due to C20orf7 mutations as a new cause of Leigh syndrome (Article) Gerards, M. Sluiter, W. Hendrickx, A. Coo, I.F.M. de Smeets, H.J. Bosch, B.J. van den Wit, L.E.A. de Calis, C. Frentzen, M. Akbari, H. Schoonderwoerd, K. Scholte, H.R. Jongbloed, R.J.	2010-08-01
Correct assay of complex I activity in human skin fibroblasts by timely addition of rotenone (Article) Wit, L.E.A. de Scholte, H.R. Sluiter, W.	2008-11-01
Functional hyperactivity of hepatic glutamate dehydrogenase as a cause of the hyperinsulinism/hyperammonemia syndrome: effect of treatment (Article) Huijmans, J.G.M. Duran, M. Klerk, J.B. de Rovers, M.J. Scholte, H.R.	2000-01-01
Biochemie van spierdydystrofie (Inaugural Lecture) Scholte, H.R.	1974-05-29

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