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protein mutation cry 1 nf 2 protein nf 2 gene clock wild-type sequence cry 2 circadian family light splice tumour exon 5 analysis point cry proteins fragment branch point sequence branch result allele activity member photoreceptor meningioma mouse ga transition patient rhythm expression acceptor genomic neurofibromatosi exons 5 addition mn 121 transcript intron position primers transcription function period nature insertion double-mutant photolyase donor locus behaviour protein kinase b detection phosphorylation absence kinase splice acceptor site nf 2 family primer probe plant blue-light receptors blot analysis donor sites cry genes 106 bp phenotype sample figure control double-mutant mice circadian rhythms transcription /translation rhythmicity wild-type mice genetic transition consensus animal disease
2 Most Recent Publications
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MammalianCry1 and Cry2 are essential for maintenance of circadian rhythms
(Letter To Editor)
Horst, G.T.J. van der Muijtjens, M. Buijs, R. Bootsma, D. Hoeijmakers, J.H.J. Yasui, A. Kobayashi, K. Takano, R. Kanno, S-I. Takao, M. Wit, J. de Verkerk, A. Eker, A.P.M. Leenen, D. van |
1999-04-15
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A G-->A transition creates a branch point sequence and activation of a cryptic exon, resulting in the hereditary disorder neurofibromatosis 2
(Article)
Klein, J.E.M.M. de Riegman, P.H.J. Bijlsma, E.K. Heldoorn, A. Muijtjens, M. Bakker, M.A. den Avezaat, C.J.J. Zwarthoff, E.C. |
1998-01-01
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