Severe hypokalaemic paralysis and rhabdomyolysis due to ingestion of liquorice
January 2005
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Chronic ingestion of liquorice induces a syndrome with findings similar to those in primary hyperaldosteronism. We describe a patient who, with a plasma K+ of 1.8 mmol/l, showed a paralysis and severe rhabdomyolysis after the habitual consumption of natural liquorice. Liquorice has become widely available as a flavouring agent in foods and drugs. It is important for physicians to keep liquorice consumption in mind as a cause for hypokalaemic paralysis and rhabdomyolysis.
Keywords
- Male
- Humans
- Middle aged
- Syndrome
- Candy
- Food Habits
- Glycyrrhetinic Acid/*toxicity
- Hypokalemia/*chemically induced/physiopathology
- Glycyrrhiza/chemistry/*toxicity
- Potassium Chloride/therapeutic use
- Rhabdomyolysis/*chemically induced/physiopathology
Automatically Extracted Terms
- liquorice
- rhabdomyolysi
- hypokalaemia
- paralysis
- liquorice consumption
- hypokalaemic paralysis
- potassium
- plasma
- consumption
- hypokalaemic
- patient
- kcl supplementation
- glycyrrhetic acid
- level
- hypertension
- cause
- plasma renin activity
- plasma k
- ingestion
- supplementation
