Wnt3a nonredundantly controls hematopoietic stem cell function and its deficiency results in complete absence of canonical Wnt signaling

2010-07-22

Blood , Volume 116 - Issue 3 p. 496- 497

Additional Metadata
Keywords	Wnt protein, Wnt1 protein, Wnt10a protein, Wnt10b protein, Wnt3 protein, Wnt3a protein, Wnt4 protein, Wnt5a protein, Wnt5b protein, Wnt7a protein, animal, cell function, cytology, genetics, hematopoiesis, hematopoietic stem cell, letter, liver, mouse, nonhuman, note, physiology, prenatal development, priority journal, protein deficiency, protein expression, signal transduction, stem cell niche, transgenic mouse, wnt3a deficiency
Persistent URL	doi.org/10.1182/blood-2010-04-282624, hdl.handle.net/1765/21105
Journal	Blood
Organisation	Erasmus MC: University Medical Center Rotterdam
Citation APA Style AAA Style APA Style Cell Style Chicago Style Harvard Style IEEE Style MLA Style Nature Style Vancouver Style American-Institute-of-Physics Style Council-of-Science-Editors Style BibTex Format Endnote Format RIS Format CSL Format DOIs only Format	Luis, T., Naber, B., Fibbe, W., van Dongen, J., & Staal, F. (2010). Wnt3a nonredundantly controls hematopoietic stem cell function and its deficiency results in complete absence of canonical Wnt signaling. Blood, 116(3), 496–497. doi:10.1182/blood-2010-04-282624

Publication