Complete ventricular septal defects (VSD) can be congenital (estimated prevalence 0.5% in live births) (Roguin N, et al. High prevalence of muscular ventricular septal defect in neonates. J Am Coll Cardiol 1995;26:1545-1548) or may be a complication of acute myocardial infarction (estimated incidence in the era of thrombolysis 0.2%) [Crenshaw BS, et al. Risk factors, angiographic patterns, and outcomes in patients with ventricular septal defect complicating acute myocardial infarction. GUSTO-I (Global Utilization of Streptokinase and TPA for Occluded Coronary Arteries) Trial Investigators. Circulation 2000;101:27-32]. In this paper, we report two unique cases of partial VSD.

Ventricular septal defect
dx.doi.org/10.1093/ejechocard/jem068, hdl.handle.net/1765/30418
European Journal of Echocardiography
Free full text at PubMed
Erasmus MC: University Medical Center Rotterdam

Pujol, E, Morales, M, Roelandt, J.R.T.C, Pérez, M.J, Masià, R, Sala, J, & Geleijnse, M.L. (2008). Partial ventricular septal defect (Pacman® Heart). European Journal of Echocardiography, 9(2), 316–317. doi:10.1093/ejechocard/jem068