XLA patients with BTK splice-site mutations produce low levels of wild-type BTK transcripts

Additional Metadata
Keywords	Bruton's tyrosine kinase (BTK), Genotype-phenotype, Real-time quantitative (RQ)-PCR, Splice-site mutation, X-linked agammaglobulinemia (XLA)
Persistent URL	dx.doi.org/10.1023/A:1019982206951, hdl.handle.net/1765/65143
Journal	Journal of Clinical Immunology
Organisation	Immunology
Citation APA Style AAA Style APA Style Cell Style Chicago Style Harvard Style IEEE Style MLA Style Nature Style Vancouver Style American-Institute-of-Physics Style Council-of-Science-Editors Style BibTex Format Endnote Format RIS Format CSL Format DOIs only Format	Noordzij, J.G, de Bruin-Versteeg, S, Hartwig, N.G, Weemaes, C.M.R, Gerritsen, E.J.A, Bernatowska, E, … van Dongen, J.J.M. (2002). XLA patients with BTK splice-site mutations produce low levels of wild-type BTK transcripts. Journal of Clinical Immunology, 22(5), 306–318. doi:10.1023/A:1019982206951

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