XLA patients with BTK splice-site mutations produce low levels of wild-type BTK transcripts

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Keywords	Bruton's tyrosine kinase (BTK), Genotype-phenotype, Real-time quantitative (RQ)-PCR, Splice-site mutation, X-linked agammaglobulinemia (XLA)
Persistent URL	doi.org/10.1023/A:1019982206951, hdl.handle.net/1765/65143
Journal	Journal of Clinical Immunology
Organisation	Department of Immunology
Citation APA Style AAA Style APA Style Cell Style Chicago Style Harvard Style IEEE Style MLA Style Nature Style Vancouver Style American-Institute-of-Physics Style Council-of-Science-Editors Style BibTex Format Endnote Format RIS Format CSL Format DOIs only Format	Noordzij, J.G, de Bruin-Versteeg, S, Hartwig, N.G, Weemaes, C.M.R, Gerritsen, E.J.A, Bernatowska, E, … van Dongen, J.J.M. (2002). XLA patients with BTK splice-site mutations produce low levels of wild-type BTK transcripts. Journal of Clinical Immunology, 22(5), 306–318. doi:10.1023/A:1019982206951

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