The tau gene in progressive supranuclear palsy: Exclusion of mutations in coding exons and exon 10 splice sites, and identification of a new intronic variant of the disease-associated H1 haplotype in Italian cases

Additional Metadata
Keywords	+39ΔG variant, Exon 4, Progressive supranuclear palsy, R406W mutation, Sequencing, Splicing effect, Tau gene
Persistent URL	doi.org/10.1016/S0304-3940(99)00669-2, hdl.handle.net/1765/67834
Journal	Neuroscience Letters
Organisation	Department of Clinical Genetics
Citation APA APA Style APA-ALL Style AAA Style Cell Style Chicago Style Harvard Style IEEE Style MLA Style Nature Style Vancouver Style American-Institute-of-Physics Style Council-of-Science-Editors Style BibTex Format Endnote Format RIS Format CSL Format DOIs only Format	Bonifati, V., Joosse, M., Nicholl, D., Vanacore, N., Bennett, P., Rizzu, P., Fabbrini, G., Marconi, R., Colosimo, C., Locuratolo, N., Stocchi, F., Bonuccelli, U., de Mari, M., Wenning, G., Vieregge, P., Oostra, B., Meco, G.& Heutink, P. (1999). The tau gene in progressive supranuclear palsy: Exclusion of mutations in coding exons and exon 10 splice sites, and identification of a new intronic variant of the disease-associated H1 haplotype in Italian cases. Neuroscience Letters, 274(1), 61–65.https://doi.org/10.1016/S0304-3940(99)00669-2

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