Genomic acute myeloid leukemia-associated inv(16)(p13q22) breakpoints are tightly clustered

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Keywords	AML, Breakpoints, CBFB, Genomic, Inv(16)(p13q22), MYH11
Persistent URL	doi.org/10.1038/sj.onc.1202321, hdl.handle.net/1765/74485
Journal	Oncogene: including Oncogene Reviews
Organisation	Department of Hematology
Citation APA APA Style APA-ALL Style AAA Style Cell Style Chicago Style Harvard Style IEEE Style MLA Style Nature Style Vancouver Style American-Institute-of-Physics Style Council-of-Science-Editors Style BibTex Format Endnote Format RIS Format CSL Format DOIs only Format	van der Reijden, B., Dauwerse, H., Giles, R. H., Jagmohan-Changur, S., Wijmenga, C., Liu, P., Smit, B., Wessels, H., Beverstock, G., Jotterand-Bellomo, M., Martinet, D., Mühlematter, D., Lafage-Pochitaloff, M., Gabert, J., Reiffers, J., Bilhou-Nabera, C., van Ommen, G., Hagemeijer, A.& Breuning, M. (1999). Genomic acute myeloid leukemia-associated inv(16)(p13q22) breakpoints are tightly clustered. Oncogene: Including Oncogene Reviews, 18(2), 543–550.https://doi.org/10.1038/sj.onc.1202321

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